In previous studies (grant et al., 1987), administration of serotonin antagonist methysergide to inhibit the increase in vascular permeability glucose increase inhibition blood pancreas. Experiments on the effects of Aloe Kidachi antidiabetic pancreas was shown to protect the back Kidachi Aloe specifically toxicity SZ (grant et al., 1993).

In this experiment, when the leakage of Evans blue intravenously test is mainly composed of a macromolecular component leaves the skin AP has more protection for the cells of the pancreatic islets that leaves the skin FD but boiled leaves the skin did not show a protective effect. Therefore, the protective effect may be associated with the polymer component is not heat resistant bark content leaf (Table 1). We (Fujita et al., 1979) detects CPase aloe leaves Kidachi, and reports the CPase fraction inhibiting increased vascular permeability associated burn injury for 20 seconds, 56 [degrees] C (Fujita et al ., 1980), as as in improving vascular permeability peritoneal inflammation in experimental models (Obata et al., 1993). For the relationship between this enzyme and the protective effect of pancreas cells clearly leaf rind AP Sephadex G-25 gel filtration chromatography, and fractionated by gel chromatography hydroxyapatite. The same fraction (leaves skin AP-F1, leaving the shell fraction AP-CPase) and CPase fraction showed the strongest protective effect (Table 3). It is recommended that the CPase closely involved in protecting cells of pancreatic islets. In addition, the skin sheet AP-F1 increases blood glucose (Doi. 5) was found to inhibit the destruction of pancreatic cells SZ delay caused. This result is consistent with previous findings (Beppu et al., 1993) Kidachi relieve diabetic condition aloe ingredient in diabetic rats SZ-induced.
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