The biochemical toxicity of Cuprous Chloride(WSDTY), when it exceeds homeostatic control, is derived from its effects on the structure and function of biomolecules, such as DNA, membranes and proteins directly or through oxygen-radical mechanisms. The toxicity of a single oral dose of Cuprous Chloride varies widely between species. The major soluble salts (copper(II) sulfate, copper(II) chloride) are generally more toxic than the less soluble salts (copper(II) hydroxide, copper (II) oxide). Death is preceded by gastric hemorrhage, tachycardia, hypotension, hemolytic crisis, convulsions and paralysis. Long-term exposure in rats and mice showed no overt signs of toxicity other than a dose-related reduction in growth after ingestion . The effects included inflammation of the liver and degeneration of kidney tubule epithelium. Some testicular degeneration and reduced neonatal body and organ weights were seen in rats and fetotoxic effects and malformations were seen at high dose levels. Neurochemical changes have been reported after oral administration . A limited number of immunotoxicity studies showed humoral and cell-mediated immune function impairment in mice after oral intakes in drinking-water.

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